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Moe-jo: you're one of the very few patients out there to have been diagnosed by an at-home sleep study. would you mind posting a separate thread on how you made the choice, whether it was your doctor's idea, how much your insurance covered, etc? i think it would be very interesting for folks on the forum to hear more about your experience.
Moe-jo said:I was diagnosed by an at-home sleep study. I wore a little machine called a WatchPAT 200. I haven't seen this mentioned elsewhere in this forum in my reading so far. I thought it was very cool.
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Alright you guys knew I had to respond to this one.
I'm happy that you had a good experience with your home study and that you've had good results.
I don't have much experience with home studies but I think the gold standard for sleep is an in-lab sleep study and an in-lab titration (the application of CPAP).
The biggest reason I'm responding is the statement that the WatchPAT 200 can detect UARS that can't be detected in the lab unless there is the application of special gauges. I don't see how one can detect the narrowing of the airway or the increased work of breathing with a snore mic, position sensor, pulse ox and "pat probe". I'm not even sure what the pat probe does.
From my research you can detect the UARS with a pressure cannula in the nose. When a study is done in-lab we put on all the things the WatchPAT has (except for perhaps the pat probe) and way more. We put on belts that measure respiratory effort, flow sensors that detect flow from nose and mouth, and a pressure cannula that detects changes in pressure from the nose. In addition there is a tech listening to what's going on in the room. So to say that a PSG wouldn't pick up UARS is a bit bogus. We just don't call it that. I should think this syndrome would present itself as either hypopneas (a reduction in airflow) or snore arousals. Both of which are addressed in the sleep study.
I think in-home sleep studies can be a valuable tool but I don't think it's a replacement for attended in-lab sleep studies. Too much room for error.
Cindy
I don't know how the WatchPAT 200 detects UARS, I simply refer to an earlier article by David E. Lawler, and his Website.
A difference could be that the PSG equipment is set to only counting apneas and Hypopneas from 10 seconds and upwards.
As far as I know the only way to detect UARS by a PSG, is combining with a PES measurement. (Pressure in the Esophageal). This is done by a little balloon in the Esophageal. This is not a procedure used with a normal PSG.
Henning
Part of my assessment letter reads:
"These are called respiratory effort related arousals (RERA's) and are picked up by the WatchPat unit in the Respiratory Disturbance Index (RDI). RDI's greater than 10 events per hour in the presence of a normal AHI constitutes a condition known as Upper Airway Resistance Syndrome (UARS)." Obviously - I didn't have a normal AHI, nor do I know how the device measured the RDI. My AHI was 20.9. My RDI was 23.7.
I am not adding this information in defense of my test or the process, or the clinic I attended. I am not entering into any debate on the subject, nor, am I offering any opinion, one way or the other. I'm just parroting this for information in case anyone is interested.
Rock, with respect to my sleep journey, I went from referral- to test- to diagnosis and treatment in fewer than four weeks. My AHI went immediately from 20.9 to a consistent number around 2, my large leak number is always 0. I know this is a lifelong process, but so far, I'm pretty happy with the pace. :-)
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