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You've got my vote! :-)
I say push a pillow over their face for a minute or so exactly four times an hour. If they complain about it, explain to them that they have no basis for complaint, since their simulated AHI has not yet reached 5.
Rock Hinkle said:Those docs view the airway as going through a stage of hypersensitivity before reaching a stage of a loss of sensitivity--kind of like how rubbing a spot on your hand will create a blister before it becomes a callus, only in this case it is the nervous system that becomes twitchy then eventually ignores the signals from the airway.
To me, that model makes sense, because it takes into consideration how the body adapts to changing circumstances--in this case, at first, sacrificing sleep in an attempt to maintain oxygenation (as in UARS) and then sacrificing oxygenation in an attempt to maintain sleep (allowing apneas), until both problems become critical (obstructive sleep apnea syndrome).
This is a good theory or model to go by. It would be hard to prove without sacrificing a few individuals. What the hell it's all in the name of science tight?! ;)
I think we should take all of the claims adjusters and put them in an isolated room for six weeks to sleep. In this room we would cut the o2 by 10% and wake them up every hour. Do you think it would make a difference?
You've got my vote! :-)
I say push a pillow over their face for a minute or so exactly four times an hour. If they complain about it, explain to them that they have no basis for complaint, since their simulated AHI has not yet reached 5.
Rock Hinkle said:Those docs view the airway as going through a stage of hypersensitivity before reaching a stage of a loss of sensitivity--kind of like how rubbing a spot on your hand will create a blister before it becomes a callus, only in this case it is the nervous system that becomes twitchy then eventually ignores the signals from the airway.
To me, that model makes sense, because it takes into consideration how the body adapts to changing circumstances--in this case, at first, sacrificing sleep in an attempt to maintain oxygenation (as in UARS) and then sacrificing oxygenation in an attempt to maintain sleep (allowing apneas), until both problems become critical (obstructive sleep apnea syndrome).
This is a good theory or model to go by. It would be hard to prove without sacrificing a few individuals. What the hell it's all in the name of science tight?! ;)
I think we should take all of the claims adjusters and put them in an isolated room for six weeks to sleep. In this room we would cut the o2 by 10% and wake them up every hour. Do you think it would make a difference?
OSA is a complete cessation in breathing. Sometimes with hypopneas that cause desats. UARS is a little more different in that it causes more arousals than o2 desats. Usually the events that are presented with UARS are Respiratory Effort Related Arousals or RERAS. A RERA is caused by flow limitation in the airway that ends with an arousal. Apneas and hypopneas are not as common with this diagnosis but can be seen. I am not an MD. It is my opinion that RERAs occur as a defense mechanism to keep an apnea, or hypopnea from occurring. Thus they prevent the desat from happening. The problem with this this type of event is the arousal. While you may not have the same health problems (depends on the extent), the arousals will cost you sleep. The loss of sleep (as I am sure that you are well aware of.) will eventually cause the problems.
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